Published in:
01-06-2019 | Neuropathic Pain | Correction
Correction to: The kinin B1 and B2 receptors and TNFR1/p55 axis on neuropathic pain in the mouse brachial plexus
Published in: Inflammopharmacology | Issue 3/2019
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Unfortunately, the Fig. 7 was wrongly downloaded in the original publication. The legend refers to the Fig. 7 presented below and it is correct in the final version of the publication:
Fig. 7
Role of BK on a genesis and b, c maintenance of mechanical hypersensitivity in mice. Mice were i.n. injected with BK and systemically treated with a, b thiorphan or c HOE 140 plus DALBK. Each group represents the mean of 5–6 animals and the vertical bars indicate the S.E.M. Different from BK group values *p < 0.05, **p < 0.01 and ***p < 0.001 (two-way ANOVA followed by Bonferroni’s post hoc test for line graph). a–c panels represent the schematic hypothesis of the drugs effect based on the results observed in panels a–c, respectively. d Thiorphan treatment blocks the conversion of BK into DABK, and also inhibits the allosteric B1R activation by CPM; e thiorphan treatment blocks the conversion of BK into DABK, and also inhibits the allosteric B1R activation by CPM even when administrated after the neuropathic-pain setting; f DALBK and HOE 140 antagonising their respective B1R and B2R. ‘B’—basal threshold; ‘NP’—neuropathic pain
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