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Translational Stroke Research
Published in: Translational Stroke Research 3/2018

01-06-2018 | Editorial

The Protein Modification and Degradation Pathways after Brain Ischemia

Authors: Tibor Kristian, Bingren Hu

Published in: Translational Stroke Research | Issue 3/2018

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Excerpt

This Special Issue of Translational Stroke Research is focused on the protein modification and degradation pathways after brain ischemia and is a continuation of a previous special issue in this journal entitled: “The protein degradation pathways after brain ischemia” [1]. Articles in this current issue summarize recent developments and understanding of protein aggregation, modification, and degradation triggered by ischemic insult. This Special Issue seeks to reiterate that disruption in cellular protein metabolism remains an important aspect of ischemic brain injury and that mechanisms of acute neuronal cell death are closely related to those of neurodegenerative disease. …
Literature
1.
2.
Yuan D, Liu Ch, Hu B. Dysfunction of membrane trafficking leads to ischemia-reperfusion injury after transient cerebral ischemia. Transl Stroke Res. 2017, in press.
3.
Yuan D, Liu Ch, Hu B. Inactivation of NSF ATPase leads to cathepsin B release after brain ischemia. Transl Stroke Res. 2017, in press.
4.
Hochrainer K. Protein modifications with ubiquitin as response to cerebral ischemia-reperfusion injury. Transl Stroke Res. 2017; in press
5.
Klimova N, Long A, Kristian T. Significance of mitochondrial protein post-translational modifications in pathophysiology of brain injury. Transl Stroke Res. 2017, in press.
6.
Dong S, Maniar S, Manole MD, Sun D. Cerebral hypoperfusion and other shared brain pathologies in ischemic stroke and Alzheimer’s disease. Transl Stroke Res. 2017, in press.
Metadata
Title
The Protein Modification and Degradation Pathways after Brain Ischemia
Authors
Tibor Kristian
Bingren Hu
Publication date
01-06-2018
Publisher
Springer US
Published in
Translational Stroke Research / Issue 3/2018
Print ISSN: 1868-4483
Electronic ISSN: 1868-601X
DOI
https://doi.org/10.1007/s12975-017-0573-z

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Translational Stroke Research 3/2018 Go to the issue

Original Article

Vasoconstriction and Impairment of Neurovascular Coupling after Subarachnoid Hemorrhage: a Descriptive Analysis of Retinal Changes

Original Article

Disease of mRNA Regulation: Relevance for Ischemic Brain Injury

Correction

Correction to: Inactivation of NSF ATPase Leads to Cathepsin B Release After Transient Cerebral Ischemia

Review Article

Dysfunction of Membrane Trafficking Leads to Ischemia-Reperfusion Injury After Transient Cerebral Ischemia

Commentary

Uncovering the Rosetta Stone: Report from the First Annual Conference on Key Elements in Translating Stroke Therapeutics from Pre-Clinical to Clinical

Original Article

Significance of Mitochondrial Protein Post-translational Modifications in Pathophysiology of Brain Injury