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Open Access 01-12-2020 | Acute Kidney Injury | Correction
Correction to: Connexin32 plays a crucial role in ROS-mediated endoplasmic reticulum stress apoptosis signaling pathway in ischemia reperfusion-induced acute kidney injury
Published in: Journal of Translational Medicine | Issue 1/2020
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Following publication of the original article [1], the authors reported errors in Fig. 5a, e. The GAPDH band in Fig. 5a was duplicated with Fig. 4b GAPDH band. In Fig. 5e, the sham picture (Cx32+/+, sham, GRP78) and the sham pictures (Cx32−/−, sham, GRP78, CHOP) were duplicated. The correct version of Fig. 5 is given below.
Fig. 5
Cx32 gene deletion mitigated I/R-induced ROS generation and ERS activation. a, b Renal I/R induced the alteration of Cx32 protein expression was detected by western blotting analysis. n = 6, *p < 0.05 vs Sham group. c Cx32 gene deletion significantly alleviated ROS production and distribution (magnification ×200). d–f Cx32 gene deletion significantly alleviated I/R (24 h)-induced GRP78, XBP1and CHOP expression increase. Changes of GRP78, XBP1and CHOP expressions were determined by western blotting analysis. Immunofluorescence staining (magnification ×400; red: GRP78 and XBP1; green, CHOP; blue: the nuclear labeled by DAPI) and immunohistochemistry (magnification ×400)
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