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Journal of Gastroenterology
Published in: Journal of Gastroenterology 6/2018

01-06-2018 | Letter to the Editor

Response to the Letter by Poddighe et al. regarding our manuscript “Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis”

Authors: Kazushige Uchida, Masato Ynagawa, Kazuichi Okazaki

Published in: Journal of Gastroenterology | Issue 6/2018

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Excerpt

We read with interest the comments by Poddighe and co-workers regarding our article “Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis” [1]. …
Literature
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Yanagawa M, Uchida K, Ando Y, et al. Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis. J Gastroenterol. 2017. (Epub ahead of print).
2.
Uchida K, Miyoshi H, Ikeura T, et al. Clinical and pathophysiological issues associated with type 1 autoimmune pancreatitis. Clin J Gastroenterol. 2016;9:7–12.CrossRefPubMed
3.
Uchida K, Tanaka T, Gershwin ME, et al. The Geoepidemiology and clinical aspects of IgG4-related disease. Semin Liver Dis. 2016;36:187–99.CrossRefPubMed
4.
Miyoshi H, Uchida K, Taniguchi T, et al. Circulating naive and CD4+ CD25high regulatory T cells in patients with autoimmune pancreatitis. Pancreas. 2008;36:133–40.CrossRefPubMed
5.
Kusuda T, Uchida K, Miyoshi H, et al. Involvement of inducible costimulator- and interleukin 10-positive regulatory T cells in the development of IgG4-related autoimmune pancreatitis. Pancreas. 2011;40:1120–30.CrossRefPubMed
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Sumimoto K, Uchida K, Kusuda T, et al. The role of CD19+ CD24high CD38high and CD19+ CD24high CD27+ regulatory B cells in patients with type 1 autoimmune pancreatitis. Pancreatology. 2014;14:193–200.CrossRefPubMed
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Fukui Y, Uchida K, Sakaguchi Y, Fukui T, et al. Possible involvement of Toll-like receptor 7 in the development of type 1 autoimmune pancreatitis. J Gastroenterol. 2015;50:435–44.CrossRefPubMed
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Ehrlich P. Beiträge zur Kenntniss der granulirten Bindegewebszellen und der eosinophilen Leukocythen. Arch Anat Physiol (Leipzig). 1879;3:166.
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Yamanishi Y, Miyake K, Iki M, et al. Recent advances in understanding basophil-mediated Th2 immune responses. Immunol Rev. 2017;278:237–45.CrossRefPubMed
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Watanabe T, Yamashita K, Sakurai T, et al. Toll-like receptor activation in basophils contributes to the development of IgG4-related disease. J Gastroenterol. 2013;48:247–53.CrossRefPubMed
Metadata
Title
Response to the Letter by Poddighe et al. regarding our manuscript “Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis”
Authors
Kazushige Uchida
Masato Ynagawa
Kazuichi Okazaki
Publication date
01-06-2018
Publisher
Springer Japan
Published in
Journal of Gastroenterology / Issue 6/2018
Print ISSN: 0944-1174
Electronic ISSN: 1435-5922
DOI
https://doi.org/10.1007/s00535-018-1460-4

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