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Critical Care
Published in: Critical Care 1/2010

01-03-2010 | Poster presentation

Acetazolamide-induced cerebrovascular reactivity is impaired in sepsis-associated encephalopathy

Authors: S Szatmari, T Végh, C Molnár, C Antek, B Fülesdi

Published in: Critical Care | Special Issue 1/2010

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Excerpt

The pathophysiology of sepsis-associated encephalopathy (SAE) is not entirely clear. One of the possible underlying mechanisms is the alteration of the cerebral microvascular function induced by the systemic inflammation. The aim of the present work was to test whether cerebral vasomotor reactivity is impaired in patients with SAE. …
Literature
1.
Wilson JX, Young GB: Sepsis-associated encephalopathy: evolving concepts. Can J Neurol Sci 2003, 30: 98-105.
2.
Matta BF, Stow PJ: Sepsis-induced vasoparalysis does not involve the cerebral vasculature: indirect evidence from autoregulation and carbon dioxide reactivity studies. Br J Anaesth 1996, 76: 790-794.CrossRef
3.
Settakis G, Molnár C, Kerényi L, et al.: Acetazolamide as a vasodilatory stimulus in cerebrovascular diseases and in conditions affecting the cerebral vasculature. Eur J Neurol 2003, 10: 609-620. 10.1046/j.1468-1331.2003.00675.xCrossRef
Metadata
Title
Acetazolamide-induced cerebrovascular reactivity is impaired in sepsis-associated encephalopathy
Authors
S Szatmari
T Végh
C Molnár
C Antek
B Fülesdi
Publication date
01-03-2010
Publisher
BioMed Central
Published in
Critical Care / Issue Special Issue 1/2010
Electronic ISSN: 1364-8535
DOI
https://doi.org/10.1186/cc8567

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