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Critical Care
Published in: Critical Care 2/2013

01-04-2013 | Letter

Tissue hypoxia during acute hemorrhage

Author: Philippe Haouzi

Published in: Critical Care | Issue 2/2013

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Excerpt

Calzia and colleagues argue [1] that tissue hypoxia in a rat model of hemorrhage that led to an oxygen deficit of 120 ml/kg with hyperlactacidemia [2] may be modest, and may not affect hydrogen sulfide oxidation since '... the arterial oxygen partial pressure was still normal ...' [1]. This contention requires clarification. …
Literature
1.
Calzia E, Radermacher P, Olson KR: Endogenous H 2 S in hemorrhagic shock: innocent bystander or central player? Crit Care 2012, 16: 183. 10.1186/cc11833PubMedCentralCrossRefPubMed
2.
Van De Louw A, Haouzi P: Oxygen deficit and H 2 S in hemorrhagic shock in rats. Crit Care 2012, 16: 178. 10.1186/cc11834CrossRef
3.
Haouzi P, Van de Louw A: Uncoupling mitochondrial activity maintains body VO 2 during hemorrhage-induced O 2 deficit in the anesthetized rat. Respir Physiol Neurobiol 2013, 186: 87-94. 10.1016/j.resp.2012.12.006CrossRefPubMed
4.
Vincent JL, De Backer D: Oxygen transport - the oxygen delivery controversy. Intensive Care Med 2004, 30: 1990-1996. 10.1007/s00134-004-2384-4CrossRefPubMed
5.
Haouzi P: Murine models in critical care research. Crit Care Med 2011, 39: 2290-2293. 10.1097/CCM.0b013e3182227550CrossRefPubMed
Metadata
Title
Tissue hypoxia during acute hemorrhage
Author
Philippe Haouzi
Publication date
01-04-2013
Publisher
BioMed Central
Published in
Critical Care / Issue 2/2013
Electronic ISSN: 1364-8535
DOI
https://doi.org/10.1186/cc12519

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