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International Orthopaedics

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01-03-2014 | Letter to the Editor

Comment on Park et al.: High glucose-induced oxidative stress promotes autophagy through mitochondrial damage in rat notochordal cells

Authors: Libo Jiang, Fenglai Yuan, Jian Dong

Published in: International Orthopaedics | Issue 3/2014

Excerpt

Dear Editor, …

Park EY, Park JB (2013) High glucose-induced oxidative stress promotes autophagy through mitochondrial damage in rat notochordal cells. Int Orthop 37(12):2507–2514. doi:10.1007/s00264-013-2037-8

Jiang L, Zhang X, Zheng X, Ru A, Ni X, Wu Y, Tian N, Huang Y, Xue E, Wang X, Xu H (2013) Apoptosis, senescence, and autophagy in rat nucleus pulposus cells: Implications for diabetic intervertebral disc degeneration. J Orthop Res 31:692–702. doi:10.1002/jor.22289 PubMedCrossRef

Park EY, Park JB (2013) Dose- and time-dependent effect of high glucose concentration on viability of notochordal cells and expression of matrix degrading and fibrotic enzymes. Int Orthop 37:1179–1186. doi:10.1007/s00264-013-1836-2 PubMedCrossRef

Kroemer G, Levine B (2008) Autophagic cell death: the story of a misnomer. Nat Rev Mol Cell Biol 9:1004–1010. doi:10.1038/nrm2529 PubMedCentralPubMedCrossRef

Kim JH, Deasy BM, Seo HY, Studer RK, Vo NV, Georgescu HI, Sowa GA, Kang JD (2009) Differentiation of intervertebral notochordal cells through live automated cell imaging system in vitro. Spine 34:2486–2493. doi:10.1097/BRS.0b013e3181b26ed1, Phila Pa 1976PubMedCrossRef

Title: Comment on Park et al.: High glucose-induced oxidative stress promotes autophagy through mitochondrial damage in rat notochordal cells
Authors: Libo Jiang
Fenglai Yuan
Jian Dong
Publication date: 01-03-2014
Publisher: Springer Berlin Heidelberg
Published in: International Orthopaedics / Issue 3/2014
Print ISSN: 0341-2695
Electronic ISSN: 1432-5195
DOI: https://doi.org/10.1007/s00264-013-2223-8

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Comment on Park et al.: High glucose-induced oxidative stress promotes autophagy through mitochondrial damage in rat notochordal cells

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