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Alzheimer's Research & Therapy

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Open Access 01-12-2023 | Amyloidosis | Retraction Note

Retraction Note: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer’s amyloidosis

Authors: Panchanan Maiti, Zackary Bowers, Ali Bourcier-Schultz, Jarod Morse, Gary L. Dunbar

Published in: Alzheimer's Research & Therapy | Issue 1/2023

Excerpt

Retraction Note: Alz Res Ther 13, 37 (2021) …

Maiti P, Bowers Z, Bourcier-Schultz A, et al. Correction to: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer’s amyloidosis. Alz Res Ther. 2022;14:42. https://doi.org/10.1186/s13195-022-00980-2.CrossRef

Maiti P, Manna J, Burch ZN, et al. Ameliorative Properties of Boronic Compounds in In Vitro and In Vivo Models of Alzheimer’s Disease. Int J Mol Sci. 2020;21(18):6664. https://doi.org/10.3390/ijms21186664.CrossRefPubMedPubMedCentral

Maiti P, Paladugu L, Dunbar GL. RETRACTED ARTICLE: Solid lipid curcumin particles provide greater anti-amyloid, anti-inflammatory and neuroprotective effects than curcumin in the 5xFAD mouse model of Alzheimer’s disease. BMC Neurosci. 2018;19:7. https://doi.org/10.1186/s12868-018-0406-3.CrossRefPubMedPubMedCentral

Title: Retraction Note: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer’s amyloidosis
Authors: Panchanan Maiti
Zackary Bowers
Ali Bourcier-Schultz
Jarod Morse
Gary L. Dunbar
Publication date: 01-12-2023
Publisher: BioMed Central
Keyword: Amyloidosis
Published in: Alzheimer's Research & Therapy / Issue 1/2023
Electronic ISSN: 1758-9193
DOI: https://doi.org/10.1186/s13195-023-01313-7

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Retraction Note: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer’s amyloidosis

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